Clinical index to Albert’s Cell biology

Note: This index will not be examined on the cell biology course.

• AIDS:
  • treatment p. 1477
  • use of CD4 and chemokine receptors: p.1550, 1581
  • AIDS virus (HIV) cell entry: p. 764, 765, 1505, 1506F, 1507
  • drug resistance: p. 1520-1521, 1522
  • HIV genome: p. 1498F
• Alzheimer’s disease: p. 397, 1001, 1021
• Amylotrophiclateral sclerosis (Lou Gehrig’s disease): p. 987
• Anthrax: p. 1492-1493, 1493F
• Anemia: p. 452, 646
• Arthritis, cronic infections: p. 1534
• Asthma:
  • allergic: p. 1556
  • chronic: p. 1534, 1534F
• Ataxia-telangiectasia(AT): p. 295T, 304, 1106
• Atherosclerosis:p. 791-792, 1500

• Bladder infection: p. 1502, 1503F
• Bubonic plague ( Y ersinia pestis):
  • evolution: p. 1520
  • influence on carrier: p. 1502,1502F

• Cancer:chapter 20, p. 1205-1267
• Cardiomyopathy:p. 103
• Chaga’s disease (trypanosomae cruzi):-invasion strategy p. 1509
• Charcot-Marie-Toothdisease: p. 1048
• Cholera:- p. 1491-1492, 1492F
• Creutzfeldt-Jakob's disease: p. 397-398
  • more about prions, BSE, scrapie: p. 1499-1498
• Cystic fibrosis: p. 666, 768

• Dyskeratosis congenita: p. 294'

• Enteritisin children (EPEC): p. 1504, 1504F
• Epidermolysis bullosa simplex: p. 986, 986F, 1005-1006

• Food poisoning:
  • Listeria monocytogenes:
    • actin-based movement:1514-1516, 1515F, 1516F
    • strategy to escape host cellmembrane traffic: p. 1511-1512, 1511F
    • destruction of phagosomal membrane p. 1512-1513, 1512F
    • invasion of host cells p.1508, 1512F
  • Salmonella enterica:
    • invasion of host cell: p.1508,1509F
    • spread and transport , 1518,1533,
    • strategy to escape host cell membrane traffic:p. 1511F, 1512, 1512F

• Hay fever: p. 1556
• Histoplasmosis (Darling’s disease): p. 1494,1494F
• Hives: p.1556
• Huntington’s disease: p. 397

• Influenza viruses: p. 1521
  • microtubule-based movement in axons: p. 1517
  • cell entry: p. 1507, 1506F
  • genome re-shuffling, pandemics: p. 1521, 1522F

• Legionnaire’s disease (Legionella pneumophilia): p. 1508, 1508F
  • strategy to escape host cell membrane traffic: p. 1511F, 1512-1513,
• Leprosy (Mycobacterium leprae): p. 1593

• Malaria (Plasmodium falciparum): p. 1494-1495, 1519
  • chloroquine resistance: p. 666
  • life cycle: p. 1495F, 1501-1502
  • MHC variant: p. 1588
• MERRF syndrome (myoclonic epilepsy with ragged red fibers): p. 866
• Multiple sclerosis 678, 680

• Myasthenia gravis: p.

• Obesity: p. 1475-1476

• Septic shock: p. 1534
• Sleeping sickness (T rypanosoma brucei):
  • antigenic variation: p. 1519,1519F
  • influence on carrier: p. 1502
  • trans-splicing: p. 354

• Stomach ulcers (Helicobacter pylori): p. 1503

• Thalassemia (an inherited form of anemia):
  • gene deletions: p. 288, 290F
  • LCR deletion: p. 452
  • Abnormal mRNA processing: p.355F

• Toxoplasmosis (Toxoplasma gondii):
  • infection mechanism: p. 1509,1510F
  • strategy to escape host cell membrane traffic: p.1512

• Tuberculosis (M ycobacterium tuberculosis): p.1488, 1507-150

• Whooping cough (B ordetella pertussis): p